NSC205 - Cellular and General Pathology, NOUN TMA Solutions

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Adele
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NSC205 - Cellular and General Pathology, NOUN TMA Solutions

Postby Adele » Sat Jul 21, 2018 5:07 pm

NSC205 - Cellular and General Pathology, NOUN TMA Solutions


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Q21 Granulomatous inflammation is a distinctive pattern of infection with ____________.

Tuberculosis

Q22 Ischemia refers to reduced supply of ___________ to tissues.

Oxygen and Nu trients

Q23 Hypoxia refers to reduced ________________ availability.

Oxygen

Q24 A special form of necrosis usually seen in immune reactions and deposition of complexes of antigens and antibodies in the walls of arteries describes _______________ necrosis.

Fib rinoid

Q25 Focal areas of fat destruction, typically resulting from release of activated pancreatic lipases into the substance of the pancreas and the peritoneal cavity describes ________________ necrosis.

Fat

Q26 A collection of fragmented or lysed cells and amorphous granular debris enclosed within a distinctive inflammatory border is characteristic of a focus of inflammation known as a __________________.

Granuloma

Q27 A form of necrosis typically encountered most often in foci of tuberculous infection is
________________ necrosis.

Caseous

Q28 A limb, generally the lower leg that has lost its blood supply and has undergone necrosis involving multiple tissue planes typically is called ______________ necrosis.

Coagulative

Q29 A non-specific pattern of cell death seen in focal bacterial or occasionally fungal infections, because microbes stimulate the accumulation is called ________________ necrosis.

Gangrenous

Q30 A phenomenon characterized by digestion of the dead cells, resulting in transformation of the tissue into a liquid viscous mass is called ____________________ necrosis.

Liquefactive

Q31 A phenomenon in which the architecture of dead tissues is preserved for a span of at least some days is called __________________ necrosis.

Coagulative

Q32 Granulomatous inflammation is a distinctive pattern of ____________ inflammation.

Chron ic

Q33 Proliferation of small blood vessels in chronic inflammation is called _____________.

Angioge nesis

Q34 Healing by connective tissue replacement of damaged tissue is called __________________.

Fibrosis

Q35 If necrotic cells and cellular debris are not promptly destroyed and reabsorbed, they tend to attract calcium salts and other minerals; a phenomenon is called dystrophic _______________________.

Calficifaction

Q36 The process in which a cell eats its own contents is called ____________________.

Necrosis
Apoptosi s
Cell death
***None of th e Options

Q37 The morphologic appearance of ____________ is the result of denaturation of intracellular proteins and enzymatic digestion of the lethally injured cell.

Necrosis


Q38 The basic pattern of cell death are ____________.

Necrosis
Apoptosi s
***Options A and B
None of the Optio ns

Q39 Nuclear features of necrosis include all but ________________________.

Karyonosis

Q40 Down syndrome is caused by a _______________ anomaly.

Chromosomal






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Adele
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Re: NSC205 - Cellular and General Pathology, NOUN TMA Solutions

Postby Adele » Sat Jul 21, 2018 5:09 pm

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Q41 Healing by connective tissue replacement of damaged tissue is called __________________,
Fibrosis

Q42 An excess of fluid in the interstitial tissue or serous cavities denotes ________________,

Exudation
Transudati on
Edema forma tion
***All of the above

Q43 A ______________ is essentially an ultrafiltrate of blood plasma that results from osmotic or hydrostatic imbalance across the vessel wall without an increase in vascular permeability.

Transuda te

Q44 Exudation implies _____________ permeability of small blood vessels in an area of injury.

Increase d

Q45 The injurious immune responses herald by excessive reactions against environmental substances or microbes describes ____________________,

Hypersensitivity rea ction

Q46 The injurious immune responses directed against self antigens describe _____________,

Autoimmune diseases

Q47 Leukocytes in chronic inflammation predominantly involve lymphocytes and ____________,

Macrophage s

Q48 Chronic inflammation may parades _______________,

Proliferation of blood vessels
Fibrosis
Tissue d estruction
***All of the above

Q49 Emigration of leukocytes in acute inflammation predominantly involves neutrophils also called ____________________ leukocytes.

Polymorphon uclear

Q50 The main characteristics of acute inflammation are _____________________,

Exudation of fluid and plasma proteins
Emigration of leukocytes
Rapid in onset
***All of the abov e

Q51 Inflammation is a complex reaction in tissues that consists mainly of vascular and
_________________ reactions.

Cellular

Q52 A fundamentally protective response, designed to rid the organism of both the initial cause of cell __________________,

Injury

Q53 The host response that gets rid of damaged or necrotic tissues and foreign invaders, such as microbes is essential called _____________________,

Inflammat ion

Q54 The stages of progressive impairment following different types of insults may include ____________,

Adaptation
Reversible injury
Cell death
***All of the above

Q55 Irreversible injury of the cell ultimately results in cell___________,

Cell Dea th

Q56 The adaptive response may consist of a change in the phenotype of cells is called ______________,

Metaplas ia

Q57 The adaptive response may consist of decrease in the size and metabolic activity of cells is called __________________,

Atrophy

Q58 The adaptive response may consist of an increase in their number is called ___________,

Hyperplasia

Q59 The adaptive response may consist of an increase in the size of cells _______________,

Hypertrophy

Q60 The reversible functional and structural response to more severe physiologic stresses and some pathologic stimuli is called cellular ___________________,

Adaptation





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Adele
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Re: NSC205 - Cellular and General Pathology, NOUN TMA Solutions

Postby Adele » Sat Jul 21, 2018 5:10 pm

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Q1 The escape of fluid, proteins, and blood cells from the vascular system into the interstitial tissue or body cavities is known as ________________.

Exudation
Transudati on
Edema forma tion
***All of the above


Q2 Stimulus for Acute Inflammation may include _____________________.

Infections
Microbial toxins
Tissue necrosis
***All of the above

Q3 Leukocytes in chronic inflammation predominantly involve lymphocytes and ____________.

Macrophage s

Q4 Types of Inflammation may be _______________.

Acute
Chron ic
***All of th e above
None of the abov e

Q5 The host response that gets rid of damaged or necrotic tissues and foreign invaders, such as microbes is essential called _____________________.

Inflammat ion

Q6 Irreversible injury of the cell ultimately results in cell___________.

Death

Q7 The morphologic appearance of ____________ is the result of denaturation of intracellular proteins and enzymatic digestion of the lethally injured cell.

Necrosis

Q8 The appearance of lipid vacuoles in the cytoplasm, seen mainly in hypoxic, toxic or metabolic injuries in cells involved in and dependent on fat metabolism, such as hepatocytes and myocardial cells is called ________________________.


Fatty change

Q9 Inability of cells to maintain ionic and fluid homeostasis and is the result of failure of energydependent ion pumps in the plasma membrane describes _____________________.

Cellular swelling

Q10 Nuclear features of necrosis include all but ________________________.

Karyonosi s

Q11 Hypoxia causes cell injury by reducing _____________ oxidative respiration.

Aerobic

Q12 Necrosis is always a _______________ process.

Pathologic

Q13 Characteristics of apoptosis include all but _____________________.

cellular contents leak out

Q14 Characteristics of necrosis include all but _____________________.

Nuclear dissolution

Q15 A cellular change caused by changes in ion concentrations and water influx is called _____________.

Cellular swelling

Q16 Which is not consistent with the hallmark of reversible injury is __________________.

Cell death

Q17 Mild forms of injury in which the functional and morphologic cellular changes are reversible if the damaging stimulus is removed is called ______________ cell injury.

Reversible

Q18 The systemic changes associated with acute inflammation are collectively called the
______________________.

Acute-phase response
Systemic inflammator y response syndrome
***A and B alone

Q19 37. Granulomatous inflammation is a distinctive pattern of ____________ inflammation.

Chron ic

Q20 Proliferation of small blood vessels in chronic inflammation is called _____________.

Angioge nesis




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Adele
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Re: NSC205 - Cellular and General Pathology, NOUN TMA Solutions

Postby Adele » Sat Jul 21, 2018 5:12 pm

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Q61 A fifth clinical sign of inflammation, _____________________, was added by Rudolf Virchow in the 19th century.

Funct io laesa

Q62 The normal cell is able to handle physiologic demands, maintaining a steady state called
____________________.

Homeostasis

Q63 Hypoxia refers to reduced ________________ availability.

Oxygen

Q64 Ischemia refers to reduced supply of ___________ to tissues.

Carbondioxide and Oxygen

Q65 The process in which a cell eats its own contents is called ____________________.

Autophagy

Q66 The morphologic appearance of ____________ is the result of denaturation of intracellular proteins and enzymatic digestion of the lethally injured cell.

Necrosis

Q67 Granulomatous inflammation is a distinctive pattern of ____________ inflammation.

Chron ic

Q68 A purulent exudate is an inflammatory exudate rich in _______________________.

Debris of dead ce lls

Q69 An excess of fluid in the interstitial tissue or serous cavities denotes ________________.

Exudation
Transudati on
Edema forma tion
***All of the above

Q70 A transudate is a fluid with high _______________________.

Albumin conten t

Q71 A ______________ is essentially an ultrafiltrate of blood plasma that results from osmotic or hydrostatic imbalance across the vessel wall without an increase in vascular permeability.

Transuda te

Q72 Exudation implies _____________ permeability of small blood vessels in an area of injury.

Increased

Q73 An exudate is an extravascular fluid that has ________________.

High protein concentration
Cellular debris
High specific g ravity
***All of the above

Q74 Calor, one of the cardinal signs of inflammation, describe ________________.

Heat

Q75 Tumor, one of the cardinal signs of inflammation, describes ________________.

Swel ling

Q76 Dolor, one of the cardinal signs of inflammation, describes ________________.

Pain

Q77 Rubor, one of the cardinal signs of inflammation, describe ________________.

Redness

Q78 Types of Inflammation may be _______________.

Acute
Chron ic
***All of th e above
None of the Opt ions

Q79 Inflammation is a complex reaction in tissues that consists mainly of vascular and
_________________ reactions.

Cellular

Q80 The host response that gets rid of damaged or necrotic tissues and foreign invaders, such as microbes is essential called _____________________.

Inflammat ion




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